A theoretical study of mass transfer between tissue and blood at the apex of a branched blood vessel

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dc.contributor.authorDavidson, MRen_AU
dc.date.accessioned2007-11-22T04:27:12Zen_AU
dc.date.accessioned2010-04-30T04:38:10Zen_AU
dc.date.available2007-11-22T04:27:12Zen_AU
dc.date.available2010-04-30T04:38:10Zen_AU
dc.date.issued1978-01en_AU
dc.description.abstractSimplified model of diffusion of inflammatory products, controlled by shear dependent permeability of the endothelium, is proposed to account for the localisation of a medial gap (caused by tissue breakdown) at a bifurcation apex, the endothelial barrier being intact at this stage. For the later stage where the artery wall is broken, a diffusion boundary layer model is proposed which predicts that, in a particular case, the diffusion flux through the break is about 400 times larger than the flux which would exist if the wall was intact at that point, and at least 40 times the down-stream flux for a shear stress of 300 dynes/cm2.en_AU
dc.identifierAAEC-E-438en_AU
dc.identifier.citationDavidson, M. R. (1978). A theoretical study of mass transfer between tissue and blood at the apex of a branched blood vessel (AAEC/E438). Lucas Heights, NSW: Australian Atomic Energy Commission, Research Establishment.en_AU
dc.identifier.govdoc662en_AU
dc.identifier.isbn0642596409en_AU
dc.identifier.placeofpublicationLucas Heights, New South Walesen_AU
dc.identifier.urihttp://apo.ansto.gov.au/dspace/handle/10238/652en_AU
dc.language.isoen_auen_AU
dc.publisherAustralian Atomic Energy Commissionen_AU
dc.subjectArteriosclerosisen_AU
dc.subjectArteriesen_AU
dc.subjectBlooden_AU
dc.subjectEndotheliumen_AU
dc.subjectMass transferen_AU
dc.subjectDiffusionen_AU
dc.subjectParticlesen_AU
dc.titleA theoretical study of mass transfer between tissue and blood at the apex of a branched blood vesselen_AU
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